Search Results

Select result number 1
1

Amyloid-β plaque formation and reactive gliosis are required for induction of cognitive deficits in App knock-in mouse models of Alzheimer’s disease

by Sakakibara, Yasufumi, Sekiya, Michiko, Saito, Takashi, Takaomi Saido, Iijima, Koichi
Published 2019
“... overproduce Aβ without non-physiological overexpression of amyloid precursor protein (APP) provide important...”

Get access
Get access

Article in Journal/Newspaper
Select result number 2
2

Amyloid-β plaque formation and reactive gliosis are required for induction of cognitive deficits in App knock-in mouse models of Alzheimer’s disease

by Sakakibara, Yasufumi, Sekiya, Michiko, Saito, Takashi, Takaomi Saido, Iijima, Koichi
Published 2019
“... overproduce Aβ without non-physiological overexpression of amyloid precursor protein (APP) provide important...”

Get access
Get access

Article in Journal/Newspaper
Select result number 3
3

Cognitive and emotional alterations in App knock-in mouse models of Aβ amyloidosis

by Sakakibara, Yasufumi, Sekiya, Michiko, Saito, Takashi, Takaomi Saido, Iijima, Koichi
Published 2018
“... transgenic models that rely on overexpression of amyloid precursor protein (APP). In App-KI mice, three...”

Get access
Get access

Article in Journal/Newspaper
Select result number 4
4

Cognitive and emotional alterations in App knock-in mouse models of Aβ amyloidosis

by Sakakibara, Yasufumi, Sekiya, Michiko, Saito, Takashi, Takaomi Saido, Iijima, Koichi
Published 2018
“... transgenic models that rely on overexpression of amyloid precursor protein (APP). In App-KI mice, three...”

Get access
Get access

Article in Journal/Newspaper

Narrow Search